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Learned Helplessness: How the Theory Reversed

Learned helplessness began with dogs and shocks in 1967. Fifty years on, its originators concluded passivity is the default and control is what is learned.

5 min read

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Key takeaways

  • Learned helplessness is the finding that, after uncontrollable aversive events, animals and people often stop trying to escape later even when escape is possible. It began with dogs and electric shocks in 1967 and was extended to students and loud noise in 1975.
  • In 2016, two of the theory's originators concluded that the original explanation was backwards. Passivity is not learned: it is the brain's default response to prolonged aversive events, and what is learned is control.
  • Most of the evidence for the revised account comes from animal studies, so how well it describes depression in people is an open question and not a settled one.
  • The human version was reformulated in 1978 around explanations: seeing a bad outcome as internal, stable and global was proposed to make the resulting passivity broader and longer lasting.
  • In the COBRA trial of 440 adults, behavioural activation delivered by junior mental health workers was not inferior to CBT at 12 months, and whether it works by teaching control is an interpretation the trial did not test.

Learned helplessness is the finding that animals and people exposed to events they cannot control often stop trying to escape later, even when escape becomes possible. It began with dogs and electric shocks in 1967 and became one of the most influential ideas about depression. [overmier-1967-shock] Fifty years later, two of the theory’s own originators reported that the original explanation had it backwards: passivity is the brain’s default response to prolonged stress, and what has to be learned is control. [maier-2016-fifty] This guide covers what the experiments showed, what the revision changed, and what it does and does not mean for depression in people.

What is learned helplessness?

Learned helplessness is a pattern of passivity that follows experience of uncontrollable aversive events. In the original experiments, dogs given electric shocks they could not escape later failed to escape shocks in a setup where escape was possible, unlike dogs without that earlier experience. [overmier-1967-shock]

The first explanation was that the animals had learned that nothing they did mattered, so they stopped trying. A human version followed in 1975, when Hiroto and Seligman reported that college students exposed to something they could not control, such as loud noise or unsolvable problems, later did worse on a different task than students who had control or no exposure. The title of the paper, Generality of learned helplessness in man, names the claim: the effect carried across very different tasks. [hiroto-1975-generality]

Both lines of work were laboratory experiments with short exposures, and the human version was run on students, so how far they describe everyday life was always an open question.

What did the theory get wrong?

The explanation was wrong even though the behaviour was real. In 2016 Maier and Seligman reviewed fifty years of neuroscience and concluded that passivity is not learned. It is the default, unlearned response to prolonged aversive events, produced by serotonin-producing neurons in a brain region called the dorsal raphe nucleus, which inhibits escape. [maier-2016-fifty]

What is learned is control. When an animal can control the event, a region called the medial prefrontal cortex detects that control and automatically inhibits the dorsal raphe response, which rescues the passivity. [maier-2016-fifty]

Original view2016 view
What is learnedThat outcomes do not depend on what you doThat outcomes can be controlled
Where passivity comes fromLearned from uncontrollable eventsThe default response to prolonged aversive events
Where the explanation sitsA belief about uncontrollabilityBrain circuits that produce passivity and detect control

One limit matters for anyone reading this as a statement about people. Most of the evidence for the revised account comes from animal experiments, so it is a well-charted account of how the brain handles uncontrollable stress in animals and an extrapolation when applied to depression in people.

How does it relate to depression?

Learned helplessness was proposed as a model of depression because the two share passivity, and the human version was reformulated in 1978 around how people explain what happens to them. Abramson, Seligman and Teasdale proposed that when someone attributes a bad outcome to causes that are internal (“it is me”), stable (“it will last”) and global (“it affects everything”), the resulting passivity is expected to be broader and longer lasting. [abramson-1978-reformulation]

That reformulation is a theory of one route into depressive passivity, not an account of the whole condition, and the animal work does not show that depression in people works this way. Depression is diagnosed on a cluster of symptoms that last at least two weeks, and our depression guide covers how clinicians define it. If the main problem is that nothing feels rewarding, our guide to anhedonia covers that separately.

What learned helplessness is not

Learned helplessness is not a diagnosis and not a character flaw. It is a name for a pattern seen in experiments, and the 2016 revision suggests the passivity is a default response of the brain to prolonged stress rather than something a person chooses or lets themselves slide into. [maier-2016-fifty]

It also does not mean that a person who feels powerless is wrong about their situation, or that their feelings are a mistake to be corrected. Sometimes the situation really is outside someone’s control, and the research above does not address how to tell the difference. What it supports is narrower: in the animal work, detecting control is what switches the passive response off. [maier-2016-fifty]

What helps?

The best-tested approach whose logic fits the revised theory is behavioural activation: planning specific activities and doing them whether or not you feel like it. In the COBRA trial, 440 adults with depression were randomised to behavioural activation delivered by junior mental health workers with brief training or to cognitive behavioural therapy. At 12 months, behavioural activation was not inferior to CBT on depression symptoms. [richards-2016-cobra]

The fit with the theory is an interpretation, not something the trial tested. If passivity is the default and control must be detected, then repeated experience of an action having an effect is a reasonable thing to build, but the trial measured symptoms and not mechanism. Our guide to CBT for depression covers how behavioural activation works in practice, and our guide to how to deal with depression covers the wider options.

A five-minute start

Pick one small task you have been putting off, such as one email, one dish or one step outside. Start the timer and do only that. The aim is to give yourself one clear piece of evidence that something you did had an effect.

5:00

Five minutes is a starting size, not a prescribed dose. Stop when the timer ends even if you want to continue. If it felt pointless, that is a common first result, and trying the same again tomorrow is the point.

When to seek help

Speak to a doctor if low mood, loss of interest, or a feeling that nothing you do makes a difference has lasted most days for two weeks or more, or if it is affecting your work, your relationships or your sleep. Our depression self-assessment is a screener and not a diagnosis, and it can help you decide whether to have that conversation.

If you feel unable to keep yourself safe, contact your local emergency services or a crisis helpline.

How MyFreud can help

The useful question about passivity is what you actually did and what happened as a result, and that is hard to see from memory on a low day. Daily mood tracking shows the pattern between the things you did, the days you did nothing and how the day felt afterwards, which turns “nothing I do matters” into something you can check against what you recorded. It cannot diagnose depression and does not replace speaking to a doctor.

Download MyFreud and start today: App Store or Google Play.

Frequently asked questions

What is learned helplessness?

Learned helplessness is the finding that animals and people who experience uncontrollable aversive events often stop trying to escape later, even when escape becomes possible. It was first reported in dogs given electric shocks in 1967 and later in students exposed to loud noise. It is a pattern seen in experiments and not a diagnosis, and its original explanation has since been revised.

Is learned helplessness actually learned?

According to a 2016 review by two of the theory's originators, no. They concluded that passivity is the default, unlearned response to prolonged aversive events, produced by serotonin-producing neurons in a brain region called the dorsal raphe nucleus. What is learned is control, which the medial prefrontal cortex detects and uses to inhibit that passive response. Most of this evidence comes from animals.

Is learned helplessness the same as depression?

No. Learned helplessness was proposed as a model of depression because the two share passivity, but it describes one possible route and not the condition itself. Depression is diagnosed on a cluster of symptoms lasting at least two weeks, and the animal work does not show that depression in people works the way learned helplessness does.

Does learned helplessness mean I am lazy or weak?

No. It is a name for a pattern seen in experiments, and the 2016 revision suggests the passivity is a default response of the brain to prolonged stress and not a choice or a character flaw. Feeling that nothing you do makes a difference is a recognised feature of low mood and a reason to speak to a doctor, not a reason for self-blame.

How do you overcome learned helplessness?

There is no treatment named after it, but behavioural activation, which means scheduling specific activities and doing them whether or not you feel like it, is a well-tested approach whose logic fits the revised theory. In the COBRA trial of 440 adults, it was not inferior to cognitive behavioural therapy at 12 months. Whether it works by teaching control is an interpretation, and the trial did not test it.

References

  1. 1.Overmier JB, Seligman MEP ( 1967). Effects of inescapable shock upon subsequent escape and avoidance responding. Journal of Comparative and Physiological Psychology.
  2. 2.Hiroto DS, Seligman MEP ( 1975). Generality of learned helplessness in man. Journal of Personality and Social Psychology.
  3. 3.Maier SF, Seligman MEP ( 2016). Learned helplessness at fifty: insights from neuroscience. Psychological Review. doi:10.1037/rev0000033
  4. 4.Abramson LY, Seligman MEP, Teasdale JD ( 1978). Learned helplessness in humans: critique and reformulation. Journal of Abnormal Psychology. doi:10.1037/0021-843X.87.1.49
  5. 5.Richards DA, Ekers D, McMillan D, et al. ( 2016). Cost and outcome of behavioural activation versus cognitive behaviour therapy for depression (COBRA): a randomised, controlled, non-inferiority trial. The Lancet. doi:10.1016/S0140-6736(16)31140-0